Fertility Restoration After Extended Anabolic Steroid Use: A Comparison of HCG and HMG
When discussing ways to maintain testicular health, size, and fertility during a hormone regimen or therapy, HCG often tops the list of recommendations.
Its efficiency is undeniable as most individuals find success in preventing or reversing testicular shrinkage and a decline in sperm production. However, there are cases where HCG might not be potent enough.
For those who have undergone rigorous anabolic steroid regimens without incorporating HCG for extensive periods, turning to HMG might be the solution.
Distinguishing HCG from HMG
HCG, found predominantly in the urine of expecting mothers (though also present in tiny amounts in men's pituitary glands), functions as a counterpart to LH (Luteinizing Hormone).
LH is the crucial hormone stimulating testosterone production, which concurrently promotes sperm production. So, by introducing HCG, individuals can substitute their natural LH (inhibited by anabolic steroids) and reactivate testicular activity.
On the other hand, HMG, sourced from the urine of women who have undergone menopause, offers a blend of peptide hormones. Unlike HCG, which mirrors LH, HMG provides both LH and FSH.
While LH can induce sperm production by raising testosterone levels, FSH (Follicle-Stimulating Hormone) is the prime enhancer of sperm generation and fertility. Hence, for those severely affected, HMG often proves more fruitful than HCG.
The Ideal Choice and Usage Guidelines
The goal should always be to avoid the necessity of HMG. This can be achieved by proactively incorporating HCG during steroid regimens, ensuring severe suppression and sterilization never occur.
For those indifferent to fertility, this guide might not be relevant. However, if undergoing hormone treatment or rigorous anabolic regimens with future parenting aspirations, a weekly dose of around 1000iu of HCG (administered as 500iu every few days) is advised. This ensures consistent testicular activity, maintains fertility, and simplifies potential transition off hormone treatments.
One key consideration is that combining HCG with hormone treatments may elevate estradiol levels. As a preventive measure, either reducing the testosterone dose or, if absolutely essential, introducing an estradiol-controlling agent is recommended.
For individuals who have been involved in extensive hormone treatments without HCG, there's a likelihood of significant fertility reduction. It's advisable to undergo necessary medical examinations, including sperm tests, to assess fertility levels.
In the event of reduced fertility, consider introducing HCG at doses of 500iu every alternate day combined with a daily dose of Enclomiphene for around six weeks. Post this period, a follow-up sperm test is necessary. If the desired results aren't achieved, consider switching to a regimen of HMG (administered twice weekly) combined with the previously mentioned dose of Enclomiphene, bypassing the HCG.
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