Introduction:

In the bodybuilding community, discussions often revolve around the positive and detrimental effects of Anabolic Androgenic Steroids (AAS). One aspect that deserves more attention is the impact of AAS use on thyroid function. This article delves into the data regarding the intricate relationship between AAS and endogenous thyroid hormones and explores the potential benefits of incorporating low doses of exogenous thyroid hormones during AAS cycles.

The Impact of AAS on Thyroid Hormone Production:

AAS exert a multifaceted influence on the regulation of thyroid hormones within the body, with outcomes that vary across individuals. When exposed to AAS, several physiological changes occur, affecting the secretion and conversion of thyroid hormones.

  1. Suppression of Thyroid-Stimulating Hormone (TSH): AAS can suppress the secretion of Thyroid-Stimulating Hormone (TSH) from the pituitary gland, which serves as the primary stimulus for the production and release of thyroid hormones, including triiodothyronine (T3) and thyroxine (T4), from the thyroid gland. Reduced TSH secretion leads to decreased T3 and T4 levels in the blood, potentially resulting in hypothyroidism.
  2. Alteration of T4 to T3 Conversion: T4 is the primary form of thyroid hormone produced by the thyroid gland but is less biologically active than T3. The conversion of T4 to T3 is a critical step in metabolic regulation, as T3 is the active form of thyroid hormone that stimulates metabolic processes. AAS can interfere with this conversion, leading to decreased levels of active T3 in the bloodstream and a reduced metabolic rate.

It's essential to note that the impact of anabolic steroids on thyroid hormones can vary based on factors such as dosage, duration of use, genetic response, and the specific type of AAS used. Stanozolol (Winstrol), for instance, has demonstrated a more significant impact on thyroid hormones than other steroids.

These negative effects can contribute to hypothyroidism and a decreased metabolic rate, making it challenging for users to shed excess body fat.

Proposed Solution:

Considering the potential for AAS to worsen thyroid function, it makes sense for AAS users to consider incorporating T3 into their cutting cycles.

While some bodybuilders opt for high doses of T3 during cutting cycles to accelerate fat loss, this approach comes with substantial health risks, including increased heart rate and blood pressure. However, using a lower dose of T3 may suffice to counteract the negative impact of AAS on endogenous thyroid production without these additional side effects.

For advanced bodybuilders already using high doses of T3 in their cutting cycles, no further action is necessary.

For those unwilling or unprepared to take high doses of T3 but still seeking to optimize thyroid function during AAS cutting cycles, a daily dosage of 12.5 to 25mcg of T3 may be a suitable option.

It's important to note that regardless of the T3 dose used, endogenous thyroid production will be suppressed by TSH shutdown. Fortunately, no post-cycle therapy (PCT) is required for thyroid hormones, as they typically recover within a couple of weeks. However, it's advisable to exercise caution with dietary intake immediately after ceasing T3.

Conclusion:

In conclusion, AAS can negatively affect thyroid production, potentially hampering the goals of bodybuilders who rely on Performance Enhancing Drugs (PEDs). Incorporating thyroid hormones during cutting cycles can provide substantial benefits for individuals looking to optimize their PED use. Careful consideration of T3 dosage, along with an understanding of its impact on endogenous thyroid function, can help bodybuilders achieve their desired results while minimizing the associated health risks.